ARG70644

Human TGF beta 3 recombinant protein (Active) (His-tagged, C-ter)

Human TGF beta 3 recombinant protein (Active) (His-tagged, C-ter) for SDS-PAGE

Overview

Product Description E. coli expressed, His-tagged (C-ter) Active Human TGF beta 3 recombinant protein
Tested Application SDS-PAGE
Target Name TGF beta 3
Species Human
A.A. Sequence Ala301 - Ser412
Expression System E. coli
Activity Active
Activity Note Determined by its ability to inhibit IL-4-induce proliferation in HT-2 cells. The ED₅₀ for this effect is less than 50 pg/mL. The specific activity of recombinant human TGF Beta 3 is approximately 2 x 10⁷ IU/mg.
Alternate Names TGF-beta3; RNHF; LAP; ARVD; ARVD1; Transforming growth factor beta-3; TGF-beta-3

Properties

Form Powder
Purification Note Endotoxin level is less than 0.1 EU/µg of the protein, as determined by the LAL test.
Purity > 98% (by SDS-PAGE)
Buffer 20 mM sodium citrate and 0.2 M NaCl (pH 4.5)
Reconstitution It is recommended to reconstitute the lyophilized protein in sterile water to a concentration not less than 200 μg/mL and incubate the stock solution for at least 20 min at room temperature to make sure the protein is dissolved completely.
Storage Instruction For long term, lyophilized protein should be stored at -20°C or -80°C. After reconstitution, aliquot and store at -20°C or -80°C for up to one month. Storage in frost free freezers is not recommended. Avoid repeated freeze/thaw cycles. Suggest spin the vial prior to opening.
Note For laboratory research only, not for drug, diagnostic or other use.

Bioinformation

Gene Symbol TGFB3
Gene Full Name transforming growth factor, beta 3
Background This gene encodes a member of the TGF-beta family of proteins. The encoded protein is secreted and is involved in embryogenesis and cell differentiation. Defects in this gene are a cause of familial arrhythmogenic right ventricular dysplasia 1. [provided by RefSeq, Mar 2009]
Function Involved in embryogenesis and cell differentiation. [UniProt]
Cellular Localization Cytoplasm
PTM The precursor is cleaved into mature TGF-beta-3 and LAP, which remains non-covalently linked to mature TGF-beta-3 rendering it inactive.